Acute periapical which pathological changes

Early visible area of periapical periodontal ligament vascular dilatation and congestion, serous exudation, tissue edema, the development of massive Europhile exudation to continue with the inflammation, tissue necrosis liquefaction and abscess formation, and the adjacent marrow cavity expansion, resulting in the limitations of the alveolar osteomyelitis, clinically known as acute alveolar abscess. Abscess center for the collapse of the periapical tissue necrosis and dental instruments pus, surrounded by Europhiles around, visible marginal zone macrophages, occasional lymphocytes, and adjacent periodontal ligament and bone marrow cavity is also visible vascular congestion and inflammatory cell infiltration.

Acute periapical abscess often more by the position of weak bony wall of the abscess, a breakthrough near the lip, buccal gingival. In addition, the root canal thick and apical large tooth abscess by cavities, via deep periodontal pocket pus of patients with periodontitis, the latter two cases are rare. Rare case of acute periapical abscess of the maxillary molars through the maxillary sinus abscess.

Supportive periapical abscess ways: the most common acute periapical abscess pus way through the submucosal abscess, apical spongy bone to reach the alveolar bone plate, by the outer plate, ha Vladimir tube to reach the subperiosteal formation of When filling the best subperiosteal abscess. Periosteal and dense, with abscesses pressure, reached a peak of pain, often accompanied by general malaise, fever, leukocytes, regional lymph node enlargement and tenderness. Once the pus is worn to the periosteal of the submucosal pain relief.